5-FU multifocal inflammatory leukoencephalopathy and dihydropyrimidine dehydrogenase deficiency

نویسندگان

  • B. Stankoff
  • A. Tourbah
  • S. Suarez
  • E. Turell
  • J. L. Stievenart
  • C. Payan
  • A. Coutellier
  • S. Herson
  • L. Baril
  • F. Bricaire
  • V. Calvez
  • E. A. Cabanis
  • L. Lacomblez
  • C. Lubetzki
چکیده

inflammatory leukoencephalopathy and dihydropyrimidine dehydrogenase deficiency Article abstract—Multifocal inflammatory leukoencephalopathy (MIL) is a cerebral demyelinating syndrome that develops after chemotherapy with 5-fluorouracil (5-FU) and levamisole. The authors report a patient who developed MIL after 5-FU administration not in association with levamisole. She was subsequently diagnosed with partial deficiency of dihydropyrimidine dehydrogenase, an enzyme necessary for 5-FU catabolism. The authors suggest that MIL is a direct result of 5-FU chemotherapy and that patients with dihydropyrimidine dehydrogenase deficiency are at increased risk for this and other toxic effects of 5-FU.

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5-FU multifocal inflammatory leukoencephalopathy and dihydropyrimidine dehydrogenase deficiency.

Multifocal inflammatory leukoencephalopathy (MIL) is a cerebral demyelinating syndrome that develops after chemotherapy with 5-fluorouracil (5-FU) and levamisole. The authors report a patient who developed MIL after 5-FU administration not in association with levamisole. She was subsequently diagnosed with partial deficiency of dihydropyrimidine dehydrogenase, an enzyme necessary for 5-FU catab...

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تاریخ انتشار 2000